For best results, use consistently as part of your daily wellness routine to support ongoing glutathione production and antioxidant status.* Each capsule delivers 900 mg of N-acetyl-L-cysteine, an amino acid that serves as the primary building block for glutathione synthesis in the body.* NAC may be more bioavailable than supplemental glutathione itself, efficiently supporting the body's ability to rebuild its glutathione supply.* You may notice support for recovery after exercise, respiratory comfort, and overall vitality with regular use.* NAC helps rebuild glutathione stores that are depleted by oxidative stress from daily living, exercise, or environmental exposures.* Results depend on lifestyle, diet, and practitioner guidance.* N-Acetyl-L-Cysteine is designed for daily use when taken as directed.* It is gluten-free, dairy-free, soy-free, and non-GMO.* Consult your healthcare practitioner before use if you are pregnant, nursing, taking medications, or managing a health condition.* Designs for Health's N-Acetyl-L-Cysteine delivers a robust 900 mg per single capsule a clinically meaningful amount.* Rather than providing antioxidants directly, NAC supports the body's own glutathione synthesis, making it a foundational building block for the body's own antioxidant status defense system.* NAC (N-Acetyl-L-Cysteine) Key Performance Benefits* Promotes normal glutathione status Supports healthy liver function and optimal detoxification Supports antioxidant status and healthy inflammatory responses Promotes muscle function and recovery Supports brain, lung, and reproductive health What This Product Is NAC provides 900 mg of N-acetyl-L-cysteine per capsule, a unique form of the amino acid cysteine that acts as a key building block for glutathione, one of the bodys most powerful antioxidants
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After this interaction, glutathione is transformed from its reduced form to its oxidized one
Absence of glutathione peroxidase-1 exacerbates cerebral ischemia-reperfusion injury by reducing post-ischemic microvascular perfusion
Lower susceptibility of female mice to acetaminophen hepatotoxicity: role of mitochondrial glutathione, oxidant stress and c-jun N-terminal kinase
